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1967). These observations have subsequently been confirmed in quite a few research and it is now properly established that there is an in depth relationship between muscle glycogen content material and fatigue resistance, each during prolonged (greater than 1 h) and during excessive-intensity intermittent train (Pernow & Saltin, 1971; Gollnick et al. 1972; Bangsbo et al. 1992; Hargreaves et al. 1995). However, the precise mechanism linking glycogen to muscle function remains elusive. The elemental features of glycogen regulation are additionally very clearly documented in any biochemistry context, with most single enzymatic and regulatory steps nicely described. Thus, it is noteworthy that we can not explain exactly why muscle function is impaired when muscle glycogen is low and how the glycogen stores have an effect on fundamental cell function. The connection between low muscle glycogen stores and fatigue is usually considered to be explained by a compromised charge of ATP regeneration and this side has been covered in different critiques (Sahlin et al.